1d11 16 8 Search Results


number hb9849  (ATCC)
92
ATCC number hb9849
Number Hb9849, supplied by ATCC, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti tgf β  (R&D Systems)
93
R&D Systems anti tgf β
Anti Tgf β, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti tgf β antibody 1d11  (R&D Systems)
90
R&D Systems anti tgf β antibody 1d11
Anti Tgf β Antibody 1d11, supplied by R&D Systems, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti transforming growth factor β tgf β alexa fluor 700  (R&D Systems)
93
R&D Systems anti transforming growth factor β tgf β alexa fluor 700
Anti Transforming Growth Factor β Tgf β Alexa Fluor 700, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
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alexa fluor 488 conjugated anti tgf β1  (Novus Biologicals)
93
Novus Biologicals alexa fluor 488 conjugated anti tgf β1
Alexa Fluor 488 Conjugated Anti Tgf β1, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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iii isoforms  (Novus Biologicals)
92
Novus Biologicals iii isoforms
Iii Isoforms, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 92 stars, based on 1 article reviews
iii isoforms - by Bioz Stars, 2026-03
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mouse monoclonal anti tgf β antibody  (Novus Biologicals)
90
Novus Biologicals mouse monoclonal anti tgf β antibody
TNBS-treatment increased the levels of anti-inflammatory cytokines in the colon of KMO-deficient mice. A: mRNA levels of <t>TGF-β,</t> IL-10, TNF-α, IFN-γ, and IL-17 in the colons of indicated mice were determined by quantitative PCR. B–C: Serial sections of the colons from vehicle or TNBS-treated KMO +/+ mice and TNBS-treated KMO +/+ or KMO −/− mice were stained for TGF-β, IL-10, Foxp3, and DAPI (nuclei). D: The ratio of TGF-β + cells or IL-10 + cells to Foxp3 + Treg cells in ILF of TNBS-treated KMO +/+ mice or TNBS-treated KMO −/− mice. Negative controls for of TGF-β, IL-10 and Foxp3 were not stained ( Supplementary Figure 3 ). The data are represented as mean ± SE from at least four independent experiments: a P < 0.05 vs TNBS-treated KMO +/+ mice group; b P < 0.01 vs TNBS-treated KMO +/+ mice group; d P < 0.01 vs KMO −/− Vehicle group. KMO: Kynurenine 3-monooxygenase; TNBS: Trinitrobenzene sulfonic acid; TGF-β: Transforming growth factor-β; IL-10: Interleukin-10; TNF-α: Tumor necrosis factor-α; IFN-γ: Interferon-γ; IL-17: Interleukin-17; Foxp3: Forkhead boxprotein P3.
Mouse Monoclonal Anti Tgf β Antibody, supplied by Novus Biologicals, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti–tgf-β clone 1d11  (BioXell Inc)
90
BioXell Inc anti–tgf-β clone 1d11
TNBS-treatment increased the levels of anti-inflammatory cytokines in the colon of KMO-deficient mice. A: mRNA levels of <t>TGF-β,</t> IL-10, TNF-α, IFN-γ, and IL-17 in the colons of indicated mice were determined by quantitative PCR. B–C: Serial sections of the colons from vehicle or TNBS-treated KMO +/+ mice and TNBS-treated KMO +/+ or KMO −/− mice were stained for TGF-β, IL-10, Foxp3, and DAPI (nuclei). D: The ratio of TGF-β + cells or IL-10 + cells to Foxp3 + Treg cells in ILF of TNBS-treated KMO +/+ mice or TNBS-treated KMO −/− mice. Negative controls for of TGF-β, IL-10 and Foxp3 were not stained ( Supplementary Figure 3 ). The data are represented as mean ± SE from at least four independent experiments: a P < 0.05 vs TNBS-treated KMO +/+ mice group; b P < 0.01 vs TNBS-treated KMO +/+ mice group; d P < 0.01 vs KMO −/− Vehicle group. KMO: Kynurenine 3-monooxygenase; TNBS: Trinitrobenzene sulfonic acid; TGF-β: Transforming growth factor-β; IL-10: Interleukin-10; TNF-α: Tumor necrosis factor-α; IFN-γ: Interferon-γ; IL-17: Interleukin-17; Foxp3: Forkhead boxprotein P3.
Anti–Tgf β Clone 1d11, supplied by BioXell Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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tgf-β neutralizing antibody clone 1d11.16.8  (GeneTex)
90
GeneTex tgf-β neutralizing antibody clone 1d11.16.8
TNBS-treatment increased the levels of anti-inflammatory cytokines in the colon of KMO-deficient mice. A: mRNA levels of <t>TGF-β,</t> IL-10, TNF-α, IFN-γ, and IL-17 in the colons of indicated mice were determined by quantitative PCR. B–C: Serial sections of the colons from vehicle or TNBS-treated KMO +/+ mice and TNBS-treated KMO +/+ or KMO −/− mice were stained for TGF-β, IL-10, Foxp3, and DAPI (nuclei). D: The ratio of TGF-β + cells or IL-10 + cells to Foxp3 + Treg cells in ILF of TNBS-treated KMO +/+ mice or TNBS-treated KMO −/− mice. Negative controls for of TGF-β, IL-10 and Foxp3 were not stained ( Supplementary Figure 3 ). The data are represented as mean ± SE from at least four independent experiments: a P < 0.05 vs TNBS-treated KMO +/+ mice group; b P < 0.01 vs TNBS-treated KMO +/+ mice group; d P < 0.01 vs KMO −/− Vehicle group. KMO: Kynurenine 3-monooxygenase; TNBS: Trinitrobenzene sulfonic acid; TGF-β: Transforming growth factor-β; IL-10: Interleukin-10; TNF-α: Tumor necrosis factor-α; IFN-γ: Interferon-γ; IL-17: Interleukin-17; Foxp3: Forkhead boxprotein P3.
Tgf β Neutralizing Antibody Clone 1d11.16.8, supplied by GeneTex, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
tgf-β neutralizing antibody clone 1d11.16.8 - by Bioz Stars, 2026-03
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anti tgf β ab  (R&D Systems)
93
R&D Systems anti tgf β ab
TNBS-treatment increased the levels of anti-inflammatory cytokines in the colon of KMO-deficient mice. A: mRNA levels of <t>TGF-β,</t> IL-10, TNF-α, IFN-γ, and IL-17 in the colons of indicated mice were determined by quantitative PCR. B–C: Serial sections of the colons from vehicle or TNBS-treated KMO +/+ mice and TNBS-treated KMO +/+ or KMO −/− mice were stained for TGF-β, IL-10, Foxp3, and DAPI (nuclei). D: The ratio of TGF-β + cells or IL-10 + cells to Foxp3 + Treg cells in ILF of TNBS-treated KMO +/+ mice or TNBS-treated KMO −/− mice. Negative controls for of TGF-β, IL-10 and Foxp3 were not stained ( Supplementary Figure 3 ). The data are represented as mean ± SE from at least four independent experiments: a P < 0.05 vs TNBS-treated KMO +/+ mice group; b P < 0.01 vs TNBS-treated KMO +/+ mice group; d P < 0.01 vs KMO −/− Vehicle group. KMO: Kynurenine 3-monooxygenase; TNBS: Trinitrobenzene sulfonic acid; TGF-β: Transforming growth factor-β; IL-10: Interleukin-10; TNF-α: Tumor necrosis factor-α; IFN-γ: Interferon-γ; IL-17: Interleukin-17; Foxp3: Forkhead boxprotein P3.
Anti Tgf β Ab, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/anti tgf β ab/product/R&D Systems
Average 93 stars, based on 1 article reviews
anti tgf β ab - by Bioz Stars, 2026-03
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Image Search Results


TNBS-treatment increased the levels of anti-inflammatory cytokines in the colon of KMO-deficient mice. A: mRNA levels of TGF-β, IL-10, TNF-α, IFN-γ, and IL-17 in the colons of indicated mice were determined by quantitative PCR. B–C: Serial sections of the colons from vehicle or TNBS-treated KMO +/+ mice and TNBS-treated KMO +/+ or KMO −/− mice were stained for TGF-β, IL-10, Foxp3, and DAPI (nuclei). D: The ratio of TGF-β + cells or IL-10 + cells to Foxp3 + Treg cells in ILF of TNBS-treated KMO +/+ mice or TNBS-treated KMO −/− mice. Negative controls for of TGF-β, IL-10 and Foxp3 were not stained ( Supplementary Figure 3 ). The data are represented as mean ± SE from at least four independent experiments: a P < 0.05 vs TNBS-treated KMO +/+ mice group; b P < 0.01 vs TNBS-treated KMO +/+ mice group; d P < 0.01 vs KMO −/− Vehicle group. KMO: Kynurenine 3-monooxygenase; TNBS: Trinitrobenzene sulfonic acid; TGF-β: Transforming growth factor-β; IL-10: Interleukin-10; TNF-α: Tumor necrosis factor-α; IFN-γ: Interferon-γ; IL-17: Interleukin-17; Foxp3: Forkhead boxprotein P3.

Journal: World Journal of Gastroenterology

Article Title: Kynurenine plays an immunosuppressive role in 2,4,6-trinitrobenzene sulfate-induced colitis in mice

doi: 10.3748/wjg.v26.i9.918

Figure Lengend Snippet: TNBS-treatment increased the levels of anti-inflammatory cytokines in the colon of KMO-deficient mice. A: mRNA levels of TGF-β, IL-10, TNF-α, IFN-γ, and IL-17 in the colons of indicated mice were determined by quantitative PCR. B–C: Serial sections of the colons from vehicle or TNBS-treated KMO +/+ mice and TNBS-treated KMO +/+ or KMO −/− mice were stained for TGF-β, IL-10, Foxp3, and DAPI (nuclei). D: The ratio of TGF-β + cells or IL-10 + cells to Foxp3 + Treg cells in ILF of TNBS-treated KMO +/+ mice or TNBS-treated KMO −/− mice. Negative controls for of TGF-β, IL-10 and Foxp3 were not stained ( Supplementary Figure 3 ). The data are represented as mean ± SE from at least four independent experiments: a P < 0.05 vs TNBS-treated KMO +/+ mice group; b P < 0.01 vs TNBS-treated KMO +/+ mice group; d P < 0.01 vs KMO −/− Vehicle group. KMO: Kynurenine 3-monooxygenase; TNBS: Trinitrobenzene sulfonic acid; TGF-β: Transforming growth factor-β; IL-10: Interleukin-10; TNF-α: Tumor necrosis factor-α; IFN-γ: Interferon-γ; IL-17: Interleukin-17; Foxp3: Forkhead boxprotein P3.

Article Snippet: The nonspecific binding was blocked with 1% BSA in PBS or M.O.M mouse Ig blocking reagent for TGF-β staining (M.O.M kit, Vector Laboratories), and the sections were subsequently incubated with rabbit anti-KMO antibody (ab83929, Abcam) and rat monoclonal anti-F4/80 antibody (ab16911, Abcam), rabbit anti-Foxp3 antibody (ab545011, Abcam), mouse monoclonal anti-TGF-β antibody (NBP2-45137, Novus Biological, Littleton, CO), rat monoclonal anti-IL-10 antibody (MBS246583, MyBioSource, San Diego, CA), rat monoclonal anti-CD19 antibody (14-0194-82, Thermo Fisher Scientific, Tokyo, Japan) and hamster monoclonal anti-CD11c antibody (70-0114, TONBO Biosciences, San Diego, CA) in 2% BSA in PBS overnight at 4 °C.

Techniques: Real-time Polymerase Chain Reaction, Staining

Administration of Kyn improved TNBS-induced colitis. KMO +/+ and KMO −/− mice were treated with the indicated KP metabolites 12 h after TNBS injection, and the colons were collected on day 3. A: Histological inflammation scores for the colons of KP metabolite-treated mice or PBS-treated mice. B: Representative images of H&E-staining in the colon of indicated mice. C: The ratios of Foxp3 + Treg cells to total CD4 + T cells in the colons of mice. D: Serial sections of the colons of from indicated mice were stained for Foxp3 and counterstained with hematoxylin, allowing indication of Foxp3 + cells. E: The mRNA expression levels of TGF-β and IL-10 were determined by quantitative PCR. The data are represented as mean ± SE from at least four independent experiments: a P < 0.05 vs PBS-treated KMO −/− mice group; b P < 0.01 vs PBS-treated KMO +/+ mice group. KMO: Kynurenine 3-monooxygenase; TNBS: Trinitrobenzene sulfonic acid; Kyn: Kynurenine; KA: Kynurenic acid; AA: Anthranilic acid; 3-HK: 3-hydroxykynurenine; 3-HAA: 3-hydroxyanthranilic acid; TGF-β: Transforming growth factor-β; IL-10: Interleukin-10; Foxp3: Forkhead boxprotein P3.

Journal: World Journal of Gastroenterology

Article Title: Kynurenine plays an immunosuppressive role in 2,4,6-trinitrobenzene sulfate-induced colitis in mice

doi: 10.3748/wjg.v26.i9.918

Figure Lengend Snippet: Administration of Kyn improved TNBS-induced colitis. KMO +/+ and KMO −/− mice were treated with the indicated KP metabolites 12 h after TNBS injection, and the colons were collected on day 3. A: Histological inflammation scores for the colons of KP metabolite-treated mice or PBS-treated mice. B: Representative images of H&E-staining in the colon of indicated mice. C: The ratios of Foxp3 + Treg cells to total CD4 + T cells in the colons of mice. D: Serial sections of the colons of from indicated mice were stained for Foxp3 and counterstained with hematoxylin, allowing indication of Foxp3 + cells. E: The mRNA expression levels of TGF-β and IL-10 were determined by quantitative PCR. The data are represented as mean ± SE from at least four independent experiments: a P < 0.05 vs PBS-treated KMO −/− mice group; b P < 0.01 vs PBS-treated KMO +/+ mice group. KMO: Kynurenine 3-monooxygenase; TNBS: Trinitrobenzene sulfonic acid; Kyn: Kynurenine; KA: Kynurenic acid; AA: Anthranilic acid; 3-HK: 3-hydroxykynurenine; 3-HAA: 3-hydroxyanthranilic acid; TGF-β: Transforming growth factor-β; IL-10: Interleukin-10; Foxp3: Forkhead boxprotein P3.

Article Snippet: The nonspecific binding was blocked with 1% BSA in PBS or M.O.M mouse Ig blocking reagent for TGF-β staining (M.O.M kit, Vector Laboratories), and the sections were subsequently incubated with rabbit anti-KMO antibody (ab83929, Abcam) and rat monoclonal anti-F4/80 antibody (ab16911, Abcam), rabbit anti-Foxp3 antibody (ab545011, Abcam), mouse monoclonal anti-TGF-β antibody (NBP2-45137, Novus Biological, Littleton, CO), rat monoclonal anti-IL-10 antibody (MBS246583, MyBioSource, San Diego, CA), rat monoclonal anti-CD19 antibody (14-0194-82, Thermo Fisher Scientific, Tokyo, Japan) and hamster monoclonal anti-CD11c antibody (70-0114, TONBO Biosciences, San Diego, CA) in 2% BSA in PBS overnight at 4 °C.

Techniques: Injection, Staining, Expressing, Real-time Polymerase Chain Reaction